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Dementias

NICE guideline [NG97] Dementia: assessment, management and support for people living with dementia and their carers. Published: Jun 2018.

NICE CKS Dementia. Last revised: Mar 2026.

Dementia

Dementia is a progressive, irreversible clinical syndrome with a range of cognitive and behavioural symptoms and reduction in the person’s ability to carry out daily activities.

This updated UKMLA guide to dementia covers various causes of dementia including Alzheimer’s disease, vascular dementia, dementia with Lewy bodies, and frontotemporal dementia, based on NICE NG97 and NICE CKS: symptoms, diagnosis, and management.

Causes and Types

Dementia is not a single disease entity; it is an umbrella term describing a clinical syndrome that can result from several underlying causes.

Main causes of dementia:

Dementia cause / type Prevalence Description
Alzheimer’s disease 50-75% of cases Pathologically associated with:

  • Extracellular amyloid plaques, and
  • Intracellular tau-rich neurofibrillary tangles

Causing cerebral cortex atrophy

Vascular dementia 20% of cases Caused by cerebrovascular disease resulting in reduced blood supply to the brain.
Dementia with Lewy bodies 10-15% of cases Characterised by Lewy body deposition in the cortical and subcortical structures
Frontotemporal dementia 2% of cases Characterised by progressive degeneration of the frontal and/or temporal lobes

Other causes include:

  • Space-occupying lesions
  • Other neurological conditions
    • Normal pressure hydrocephalus
    • Parkinson’s disease dementia
    • Progressive supranuclear palsy
    • Huntington’s disease
    • Prion disease (e.g. CJD)
  • Metabolic conditions
    • Hypothyroidism
    • Vitamin B12 deficiency
    • Vitamin B1 (thiamine) deficiency
    • Hypercalcaemia
    • Uraemia (in renal impairment)
    • Hepatic encephalopathy
    • Adrenal insufficiency, hypopituitarism
  • Infections
    • HIV dementia
    • Tertiary syphilis

Be aware of medications that can contribute to cognitive impairment

  • Those with anticholinergic properties (e.g. TCA, anticholinergics used for overactive bladder, antihistamines, antipsychotics)
  • Benzodiazepines
  • Opioids

Risk Factors

General risk factors:

  • Older age – strongest risk factor
  • Mild cognitive impairment (~1/3 of those will develop dementia within 3 years)
  • Learning disability

Cause / type-specific risk factors:

Dementia type Risk factors
Alzheimer’s disease Genetic risk factors:

  • ApoE E4 is the major genetic susceptibility factor for late-onset Alzheimer’s disease
  • PSEN 1/2 and APP gene mutations are associated with early-onset Alzheimer’s disease

Down syndrome (trisomy 21) is a major risk factor for early-onset Alzheimer’s disease, as the APP gene is located on chromosome 21, and Down syndrome patients have 3 copies

Modifiable risk factors (~1/3 of cases may be attributable to modifiable risk factors):

  • Lower educational attainment (higher levels of education, more mentally demanding jobs, and cognitive stimulation are associated with a lower risk of developing dementia)
  • Low social engagement and support
  • Medical conditions
    • Hearing impairment (may result in cognitive decline through reduced cognitive stimulation)
    • Hypertension
    • Depression
    • Diabetes
    • Traumatic brain injury
  • Lifestyle factors
    • Obesity
    • Physical inactivity
    • Smoking
    • Excessive alcohol consumption (moderate alcohol consumption may be protective)
    • Air pollution
Vascular dementia Mainly contributed by cerebrovascular disorders

  • Large or multiple small infarcts
  • Hypertensive small vessel disease
  • Cerebral amyloid angiopathy
  • CADASIL syndrome – most common single-gene disorder causing stroke

General cardiovascular risk factors also increase risk, including:

  • Smoking
  • Hypertension
  • Diabetes
  • Concurrent cardiovascular disease (e.g. coronary artery disease, peripheral arterial disease, stroke, TIA)
Dementia with Lewy bodies
  • Older age
  • Male sex
  • Family history
Frontotemporal dementia
  • Younger age (typical onset: 60-69 y/o)
  • Family history

Clinical Features

Dementia has a gradual onset and non-specific signs and symptoms, which vary from person to person.

Non-Specific / Shared Clinical Features

Suspect dementia if ANY of the following are reported by the patient and/or family / carer:

Category Clinical features
Cognitive impairment Impairment in any of the following domains

  • Memory
    • Difficulty remembering recent events, names or conversations
    • Difficulty learning new information
    • Vague with dates or misses appointments
    • May defer to family/carers when answering questions
    • May repeat questions or stories
  • Language
    • Word-finding difficulty
    • Difficulty following or producing speech
    • Problems naming objects
    • Dysphasia may occur as dementia progresses
  • Visuospatial
    • Getting lost in familiar places
    • Difficulty judging distances
    • Problems navigating, driving or recognising routes
    • Difficulty recognising objects or faces in some cases
  • Executive function
    • Difficulty planning, organising or problem-solving
    • Difficulty in reasoning and communication
    • Difficulty making decisions
    • Poor judgement
    • Difficulty managing finances, medications, cooking or appointments
    • Difficulty carrying out multi-step tasks, e.g. dressing or preparing a meal
Behavioural and psychological symptoms of dementia (BPSD) BPSD are essentially non-cognitive symptoms

BPSD tend to fluctuate, and may last 6 months or more:

  • Behavioural symptoms
    • Disinhibition – socially or sexually inappropriate behaviour
    • Motor disturbance (e.g. wandering, restlessness, pacing, and repetitive activity)
    • Sleep disturbance or insomnia
    • Repeating phrases or questions
  • Psychological symptoms
    • Psychosis – possible delusions (which may be persecutory) and/or hallucinations (visual and auditory)
    • Depression and anxiety – the onset of depression in later life is a warning sign of dementia
    • Emotional lability (e.g. easily upset, argumentative, shout, have mood swings, and/or exhibit physically and verbally challenging behaviour)
    • Withdrawal or apathy
Difficulties with activities of daily living (ADLs) Examples include:

  • Complex household tasks (in early stages)
  • Basic ADLs may be impacted in later stages (e.g. bathing, toileting, eating, walking)

Type-Specific Clinical Features

Dementia type Specific clinical features
Alzheimer’s disease Typical presenting symptom

  • Recent memory loss
  • Loss of episodic memory (e.g. memory loss for recent events, repeated questioning, difficulty learning new information)
  • Executive function difficulty
  • Nominal dysphasia (difficulty in retrieving words, esp. nouns and verbs)

Alzheimer’s disease often co-exists with other forms of dementia such as vascular dementia.

Vascular dementia Patient would likely have a history of cardiovascular / cerebrovascular diseases (esp. stroke / TIA) and/or risk factors

  • Stepwise cognitive decline
    • Characterised by a sudden or noticeable deterioration in cognition (often after vascular events), followed by a period of relative stability
    • This contrasts with the gradual decline pattern seen in Alzheimer’s disease
  • Early executive dysfunction
  • Gait disturbances
  • Change in personality
  • Focal neurological deficit (e.g. hemiparesis, visual field defect)

Imaging may show old infarcts or small vessel disease.

Dementia with Lewy bodies Core tetrad features:

  • Fluctuating cognition
  • Visual hallucinations (tend to involve humans or animals)
  • Parkinsonism (e.g. bradykinesia, cogwheel rigidity, resting tremor)
  • REM sleep disorder

Memory impairment may not be apparent in early stages

Dementia with Lewy bodies vs Parkinson’s disease dementia

They are closely related conditions within the spectrum of Lewy body disease, a textbook rule:

  • Dementia with Lewy bodies: dementia develops before or within 1 year of parkinsonism
  • Parkinson’s disease dementia: dementia develops after 1  year of parkinsonism
Frontotemporal dementia Typical onset in 60-69 y/o with family history

  • Personality change
  • Behavioural disturbances
    • Social disinhibition (e.g. making inappropriate jokes or comments, saying rude or offensive things)
    • Sexual disinhibition (e.g. making inappropriate sexual comments, public masturbation or exposure)
    • Apathy

Other cognitive functions (such as memory and perception) may be relatively preserved

Dementia Mimics

Dementia and delirium can both present with confusion, memory problems and impaired functioning:

Feature Dementia (non-advanced cases) Delirium
Onset Gradual onset Acute / subacute onset
Course Progressive decline over months to years Fluctuating course over hours to days
Consciousness Usually normal Altered level of consciousness is common
Attention Usually preserved Impaired attention is a core feature
Orientation Usually normal Often impaired
Hallucinations Mainly in dementia with Lewy bodies Common, often visual hallucinations
Reversibility Progressive and irreversible Often reversible if the underlying cause is treated

Key distinction: dementia is a chronic progressive cognitive disorder, whereas delirium is an acute fluctuating confusional state.

To distinguish between delirium and dementia, NICE recommends using the:

  • Confusion assessment method (CAM)
  • Observational Scale of Level of Arousal (OSLA)

If not possible to tell between delirium and dementia, or delirium superimposed on dementia → treat delirium first

Depression can cause cognitive symptoms that mimic dementia, sometimes referred to as pseudodementia

Feature Dementia Depression / pseudodementia
Onset Usually gradual and progressive May be more clearly linked to a mood change or life event
Mood symptoms Possible, but cognitive symptoms are usually the main concern Low mood, anhedonia, hopelessness, poor sleep, appetite changes are often prominent
Cognitive complaints Lack of insight into the cognitive symptoms is common Patient often emphasises memory problems and may be very distressed by them
Effort during consultation / assessment Good effort, often attempts answers but may be incorrect Poor effort, may give “I don’t know” answers
Pattern of impairment Progressive decline affecting daily function Cognitive symptoms may fluctuate with mood and concentration

Late-life depression may also be a risk factor for, or early feature of, underlying dementia.

Prognosis

Dementia is a life-limiting condition, there is no treatment currently available to cure dementia or stop disease progression

Dementia and Alzheimer’s disease are the leading cause of death for women (13.4% of deaths) and 2nd leading cause of death for men (7% of deaths)

Time from presentation to death (varies by age):

  • Late 60s to early 70s = median lifespan of 7-10 years
  • 90s = median lifespan of 3 years

Investigation and Diagnosis

For a diagnosis of dementia to be made, there must be impairment:

  • In at least two of the following cognitive domains: memory, language, behaviour, visuospatial or executive function, and
  • Which causes a significant functional decline in usual activities or work, and
  • Which cannot be explained by delirium or other major psychiatric disorder

Early-onset (or young-onset) dementia is generally defined as dementia that develops <65 y/o

Mild cognitive impairment is cognitive impairment that does not fulfil the diagnostic criteria for dementia (e.g. only one cognitive domain is affected, or deficits do not significantly affect daily activities)

Primary Care

Clinical assessment
  • Focused history + collateral history (e.g. from a family member or carer), and
  • Physical examination (including neurological examination)
  • Medication review
Cognitive testing NICE recommends the following validated cognitive assessment tool

  • 10-point cognitive screener (10-CS)
  • 6-item cognitive impairment test (6CIT)
  • 6-item screener
  • Memory Impairment Screen (MIS)
  • Mini-Cog
  • Test Your Memory (TYM)
Blood tests To exclude reversible causes of cognitive decline

  • Standard bloods: FBC, CRP, ESR, U&E, LFTs
  • Calcium – to check for hypercalcaemia
  • TFT – to check for hypothyroidism
  • Serum B12 and folate – to check for deficiency
  • HbA1c

Other investigations that may be appropriate if clinically indicated include urine microscopy and culture, chest X-ray, ECG, syphilis serology, and HIV testing

If dementia is suspected and reversible causes have been investigated → refer to specialist dementia diagnostic service (e.g. memory clinic, community old age psychiatry service)

NICE did not specifically recommend the use of MoCA and MMSE which are still often used in practice. Expert summaries and reviews commented that:

  • Longer or more complex tests may not be better than shorter simpler tools in primary care.
  • MoCA is reported as not well tolerated by people with suspected dementia (ACNR Journal review) [Ref]

Secondary Care

Dementia and its subtypes can be diagnosed clinically by a specialist

Also include a test of verbal episodic memory if Alzheimer’s disease is suspected

Imaging

Offer structural imaging (usually MRI brain, alternatively CT) to rule out reversible causes of cognitive decline and assist with subtype diagnosis

  • Unless dementia is well established and the subtype is clear

Consider further imaging if there is diagnostic uncertainty for the dementia subtype (and would change management):

Suspected dementia subtype Test of choice Findings
Alzheimer’s disease Either of the following:

  • Functional brain imaging: FDG-PET or perfusion SPECT scan
  • CSF analysis for tau protein and amyloid beta
Functional brain imaging:

  • Hypometabolism in temporoparietal cortex and posterior cingulate cortex

CSF analysis:

  • ↑ Total tau protein and phosphorylated tau
  • ↓ Amyloid beta 1-42 and 1-40
Lewy body dementia
  • 1st line: DaT scan (123I‑FP‑CIT SPECT)
  • 2nd line: 123I‑MIBG cardiac scintigraphy

Do not rule out Lewy body dementia based on normal results

DaT scan

  • ↓ Dopamine transporter uptake in the basal ganglia (esp. in putamen and caudate nuclei)

Cardiac scintigraphy:

  • ↓ Uptake in myocardium (reflects reduced cardiac sympathetic innervation)
Frontotemporal dementia Functional brain imaging:

  • FDG-PET or
  • Perfusion SPECT scan
Reduced function in frontal and/or anterior temporal lobes:

  • FDG-PET: reduced glucose metabolism
  • SPECT: reduced blood flow
Vascular dementia
  • 1st line: MRI
  • 2nd line: CT
Ischaemic white matter changes:

  • Diffuse white matter hyperintensities on T2/FLAIR
  • Cortical and subcortical infarcts / lacunar infarcts
  • Cerebral atrophy (patchy)

Clarification of some confusing terms – different types of SPECT imaging.

  • SPECT is a general imaging technique
  • 123I‑FP‑CIT SPECT is a specific type of SPECT where 123I‑FP‑CIT binds to dopamine transporters thus is also called DaT scan. This is used to assess dopaminergic activity and used for Lewy body dementia and Parkinsonian syndromes
  • When NICE just mentions SPECT, they are referring to brain perfusion SPECT that uses tracers like technetium-99m which measures regional cerebral blood flow

Therefore, the SPECT mentioned in Lewy body dementia, Alzheimer’s disease and frontotemporal dementia are NOT interchangeable.

On the other note, PET scans assess glucose uptake and metabolism in tissue using 18F-FDG tracer.

Management

Conservative / General Management

Offer:

  • Range of activities to promote wellbeing tailored to the patient’s preferences
  • Group cognitive stimulation therapy (for mild to moderate dementia)

Consider the following for mild to moderate dementia:

  • Group reminiscence therapy
  • Cognitive rehabilitation or occupational therapy

Behavioural and Psychological Symptoms of Dementia (BPSD)

1st line: non-pharmacological interventions (psychosocial interventions, environmental adaptations, verbal/non-verbal de-escalation)

  • Ensure reversible triggers have been ruled out / managed (e.g., pain, infection, delirium, adverse effects of medications)

2nd line: pharmacological interventions

  • Short-term antipsychotic therapy
    • Indication
      • Unresponsive to non-pharmacological interventions AND
      • At risk of harming themselves or others, OR
      • Experiencing agitation, hallucinations, or delusions that are causing severe distress
  • Licensed options (AD/VD): haloperidol and risperidone (started under specialist supervision)

Antipsychotics carry a significant risk of cerebrovascular events (e.g. stroke) and increased mortality in people with dementia.

When used, they should be prescribed at the lowest effective dose, for the shortest possible duration, with regular review of ongoing need.

General Pharmacological Management

Pharmacological management depends on the type of dementia.

Alzheimer’s Disease

For mild to moderate disease:

  • Step 1: AChE inhibitor (donepezil / galantamine / rivastigmine)
  • Step 2: add memantine

Consider memantine monotherapy in:

  • Severe Alzheimer’s disease on presentation, or
  • Acetylcholinesterase inhibitors not appropriate

Lewy Body Dementia

NICE guideline focuses only on general dementia management and does not elaborate on a symptom-based management approach, which is particularly relevant for Lewy body dementia:

Symptom Pharmacological management
Neurocognitive (i.e. dementia) Similar to Alzheimer’s management:

  • 1st line: AChE inhibitor
  • Alternative: consider memantine
Psychiatric
  • 1st line: AChE inhibitor
  • 2nd line: 2nd-generation antipsychotics (usually quetiapine)
Motor (i.e. Parkinsonism)
  • Levodopa (cautiously)

Antipsychotics should be avoided in Lewy body dementia (and Parkinson’s disease dementia), as they can precipitate motor symptoms.

In these cases, non-pharmacological strategies and cholinesterase inhibitors should be prioritised for neuropsychiatric symptoms before considering antipsychotics.

Vascular Dementia

The mainstay of management of vascular dementia is identification and control of vascular risk factors (including hypertension, hyperlipidaemia, and diabetes)

Do NOT routinely offer AChE inhibitor or memantine

Only consider if the patient has suspected comorbid:

  • Alzheimer’s disease
  • Lewy body dementia
  • Parkinson’s disease dementia

Frontotemporal Dementia

The mainstay of management for frontotemporal dementia is symptomatic and supportive care (focused on non-pharmacological interventions and caregiver support)

Do not routinely offer AChE inhibitor or memantine

Dementia and Driving

If the patient has dementia and/or any organic syndrome affecting cognitive functioning

Licence type Recommendation
Group 1 vehicle (car / motorcycle) May be able to drive but must notify the DVLA

  • The decision on licensing is usually based on medical reports.
  • Poor short-term memory, disorientation, and lack of insight and judgement almost certainly mean no fitness to drive
Group 2 vehicle (bus / coach / lorry) Must not drive and must notify the DVLA

Licensing will be refused or revoked

References

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