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Idiopathic Intracranial Hypertension (IIH)

Mollan SP, Davies B, Silver NC, et al. Idiopathic intracranial hypertension: consensus guidelines on management. Journal of Neurology, Neurosurgery & Psychiatry 2018;89:1088-1100.

European Headache Federation guideline on idiopathic intracranial hypertension. Published: Oct 2018.

Idiopathic Intracranial Hypertension (IIH)

Idiopathic intracranial hypertension is a condition characterised by raised intracranial pressure without an identifiable structural or secondary cause.

Updated UKMLA guide to idiopathic intracranial hypertension based on European Headache Federation guideline, which covers causes, risk factors, symptoms, complications, diagnosis, and management.

Causes and Risk Factors

The exact underlying cause or pathogenesis of IIH remains unknown (thus idiopathic).

Primary risk factors: [Ref1][Ref2]

  • Obesity – most significant risk factor (90-95% of patients are obese)
  • Recent weight gain (5-15% of body weight)
    • Occurring in the period preceding symptom onset or diagnosis is a major trigger and risk factor
  • Women of childbearing age

Clinical Features

Typically presents in an obese female of childbearing age [Ref1][Ref2]

Category Clinical features
Headache Most common symptom

  • Typically progressively severe and frequent
  • The pain is often pressing or explosive with a frontal or retro-orbital location

However, the headache phenotype is highly variable, frequently mimicking a migraine

Ophthalmic features
  • Transient visual disturbances (e.g. darkening or dimming of vision)
  • Papilloedema
  • Horizontal diplopia (from CN VI palsy)

In true IIH, there should be absolutely no other cranial nerve deficits other than a potential CN VI palsy

Auditory and olfactory features
  • Pulsatile tinnitus (whooshing or humming sound that occurs with the heartbeat)
  • Altered / reduced sense of smell
Systemic features
  • Cognitive impairment (esp. impacting reaction time and processing speed)
  • Pain (back, neck, radicular)
  • Dizziness

Complications

[Ref1][Ref2]

  • Permanent visual loss – most severe complication of IIH from prolonged papilloedema
  • Chronic persisting headache – primary factor contributing to reduced QoL
    • Medication-overuse headache may also develop due to the patient frequently relying on analgesia
  • Cognitive impairment – may persist even after ICP and headache improve

Investigation and Diagnosis

Work-Up

[Ref1][Ref2]

Category Description / test
Clinical / bedside assessment
  • Comprehensive neurological and ophthalmological examination
  • Blood pressure measurement (to exclude malignant hypertension)
  • FBC to exclude severe anaemia and other secondary causes if necessary
Neuroimaging
  • Urgent MRI brain to exclude structural causes of raised ICP
    • If not available → CT
  • CT / MR venogram to exclude cerebral venous sinus thrombosis
Lumbar puncture If neuroimaging is normal → lumbar puncture

  • Opening pressure measurement
  • CSF analysis

Do not perform lumbar puncture before neuroimaging if raised ICP is suspected, due to the risk of brain herniation if there is a space-occupying lesion.

If raised ICP is due to a space-occupying lesion, removing CSF from the spinal canal during lumbar puncture can create a pressure gradient between the skull and spinal canal. This can pull brain tissue downwards and cause brain herniation, especially tonsillar herniation, which can compress the brainstem and become rapidly fatal.

Diagnostic Criteria

Friedman diagnostic criteria – ALL of the following must be met: [Ref1][Ref2]

  • Papilloedema is present
  • Normal neurological examination (except CN VI palsy)
  • Normal neuroimaging (i.e. no structural causes, meningeal enhancement, cerebral venous sinus thrombosis)
    • Typical neuroimaging findings include empty sella, flattening of the posterior globe, distension of perioptic subarachnoid space (optic nerve sheath), transverse venous sinus stenosis
  • Normal CSF analysis
  • ↑ Lumbar puncture opening pressure (≥25 cm H₂O in adults or ≥28 cm H₂O in obese children)

IIH without papilloedema is a rare variant where there is ↑ ICP but no optic disc swelling, additional diagnostic findings are necessary.

Although not included in the diagnostic criteria, it is important to exclude secondary causes of raised ICP: [Ref1][Ref2]

  • Medications
    • Antibiotics: tetracyclines, fluoroquinolones
    • Retinoids / vitamin A derivatives
    • Hormones and steroids: corticosteroid withdrawal, anabolic steroids, growth hormone, COCPs
    • Lithium
  • Venous drainage obstruction
    • Cerebral venous sinus thrombosis
    • Jugular vein thrombosis
    • SVC syndrome
    • ↑ Central venous pressure (e.g. heart failure)
  • Systemic and medical disorders
    • CKD, uraemia, OSA, COPD
    • SLE
    • Infections: HIV, Lyme disease, varicella, psittacosis, CNS infections, otitis media, mastoiditis
  • Endocrine disorder
    • Addison’s disease, adrenal insufficiency
    • Thyroid and parathyroid disorders
    • Cushing’s syndrome
  • Haematological disorders
    • Anaemia (including severe iron deficiency)
    • Thrombophilia
    • Polycythaemia vera
  • Syndromic / genetic conditions
    • Down syndrome
    • Turner syndrome
    • Craniosynostosis

Exclusion of secondary causes is especially important in those who do not fit the “obese woman of reproductive age” demographic. [Ref]

Management

[Ref1][Ref2]

Category Management
Weight loss and management For ALL patients with BMI >30 kg/m2

Bariatric surgery is increasingly considered for sustained, long-term weight loss in IIH

Medical therapy To be considered in those with active symptoms with no immediate threat to vision

  • 1st line: acetazolamide (reduces CSF secretion)
  • Topiramate (reduces CSF secretion and suppresses appetite)

However, ~50% of patients discontinue acetazolamide because of its significant side effects (like fatigue, tingling, altered taste, and depression)

Surgical intervention Surgery is ONLY indicated for those with visual disturbancespapilloedema

Options include:

  • CSF diversion with VP shunt – preferred procedure in the UK
  • Optic nerve sheath fenestration
  • Endovascular stenting – for transverse venous sinus stenosis

If vision is actively declining (fulminant IIH), a lumbar drain or serial lumbar punctures can be used as a short-term temporary measure, while awaiting definitive surgery.

Surgical interventions are NOT recommended exclusively for treating headaches due to poor outcomes, high revision rates, and risk of complications

References

Related Articles

Raised Intracranial Pressure (ICP)

Papilloedema

Cerebral Venous Sinus Thrombosis (CVST)

Migraine

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