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Allergic Rhinitis

NICE CKS Allergic rhinitis. Last revised Jan 2024.

Allergic Rhinitis

Allergic rhinitis is an IgE-mediated inflammatory condition of the nose triggered by exposure to allergens such as pollen, house dust mites, mould, or animal dander. Hay fever is a specific form of allergic rhinitis, referring to seasonal allergic rhinitis triggered by seasonal allergens such as pollen.

This updated UKMLA guide to allergic rhinitis is based on NICE CKS, which covers causes, symptoms, diagnosis, and management.

Classification

Seasonal rhinitis (hay fever): symptoms occur at the same time each year in response to a seasonal allergen

  • Most typically occurs in the summer but also depends on the pollen

Perennial rhinitis: symptoms occur throughout the year

Causes

Genetic predisposition is an important factor in allergic rhinitis development. Risk factors include:

  • Family history of atopy (esp. allergic rhinitis)
  • Food allergy in childhood

Asthma and allergic rhinitis often co-exist. Allergic rhinitis is a risk factor for the development of asthma.

Common environmental triggers:

  • Pollens (grass, tree, weed)
  • House dust mites
  • Moulds
  • Cat and dog hair

Clinical Features

Typical features:

  • Classic bilateral symptoms that develop within minutes post-allergen exposure
    • Sneezing
    • Nasal itching
    • Rhinorrhoea
    • Nasal congestion
  • Postnasal drip
  • Cough, palate itching
  • Features of chronic nasal congestion (e.g. snoring, mouth breathing, halitosis)
  • Turbinate hypertrophy

Allergic rhinitis can co-exist with the following conditions:

  • Asthma
  • Allergic conjunctivitis
  • Oral allergy syndrome
  • Chronic sinusitis and nasal polyps (as a complication)

Allergic rhinitis can cause sleep disturbances and impact quality of life, especially during the peak pollen season.

Symptoms occur following exposure to a known causative allergen

  • Tree pollens — intermittent or chronic symptoms occur from early to late spring.
  • Grass pollens — intermittent or chronic symptoms occur from late spring to early summer.
  • Weed pollens — intermittent or chronic symptoms may occur from early spring to early autumn.
  • House dust mites — symptoms are worse on waking and are present all year-round, but may peak in autumn and spring.
  • Animal dander — symptoms follow exposure to animal dander, and may be all year-round or occasional, depending on exposure.
  • Occupational — intermittent or chronic symptoms tend to improve when the person is away from work, such as weekends and holidays.

Investigation and Diagnosis

Allergic rhinitis is primarily a clinical diagnosis based on a combination of:

  • Typical history
  • Clinical examination
  • Response to 1st line treatment

Specialist may perform further testing if there is diagnostic doubt or failure to respond to treatment

  • Allergen testing
    • Skin prick testing
    • RAST (measure level of serum-specific IgE to allergens)
  • Nasal allergen challenge
  • Nasal endoscopy
  • CT

Management

General Advice / Conservative Management

  • Consider nasal irrigation with saline solution
  • Allergen avoidance (highly depends on the identified causative allergen, if any)

Specific Allergen Avoidance Techniques

Causative allergen Allergen avoidance techniques
Pollen allergy
  • Avoid walking in grassy, open spaces when the pollen count is high (particularly during the early morning and early evening)
  • Avoid drying washing outdoors when the pollen count is high
  • Keep windows shut in cars and buildings when the pollen count is high
  • Plan holidays to avoid the pollen season, where possible
  • Shower or wash hair following high pollen exposures
  • Consider the use of sunglasses (ideally wraparound) or nasal barriers (masks covering the nose and mouth or commercially available powders, balms or creams rubbed on the nose) when the pollen count is high
  • Consider monitoring the pollen count using a website
House dust mite allergy
  • Not fit mattresses, pillows, and duvets with house dust mite impermeable covers
  • Use synthetic pillows and acrylic duvets, and keep furry toys off the bed
  • Wash all bedding and furry toys at least once a week at high temperatures
  • Choose wooden or hard floor surfaces instead of carpets, if possible
  • Fit blinds that can be wiped clean instead of curtains. Surfaces should be wiped regularly with a clean, damp cloth
Animal allergy
  • Ideally not allow the animal in the house
    • If this is not acceptable or possible, advise restricting their presence to the kitchen
  • Regularly wash the animal and any surfaces they are in contact with
Occupational allergies
  • Avoid exposure to allergen completely where possible
  • If elimination or complete avoidance of the allergens is not possible, reduce exposure to both known and potentially sensitising allergens in the workplace (e.g. use latex-free gloves, wear protective clothing, or a dust mask)
  • Ensure that their work environment is adequately ventilated and/or relocate to lower exposure areas in the workplace
  • Use less hazardous chemicals, if possible and appropriate

Pharmacological Management

1st Line Therapy

  • Intranasal corticosteroids (e.g. mometasone, fluticasone), AND/OR
  • Antihistamine (intranasal or oral)
    • Intranasal: Azelastine hydrochloride is the only licensed intranasal spray
    • Oral: offer 2nd-generation antihistamines like cetirizine, loratadine, fexofenadine

If there are also eye symptoms → add antihistamine eye drops or chromone eye drops (sodium cromoglycate, nedocromil)

Other information:

Preference for treatment options
  • Children with mild and/or intermittent symptoms → antihistamines preferred (intranasal or oral)
  • Adolescents and adults with mild and/or intermittent symptoms→ no particular preference (any combination)
  • Moderate to severe symptoms (i.e. impacting quality of life) or persistent intranasal corticosteroid +/- intranasal antihistamine
What is the most effective regimen?
  • Intranasal corticosteroid + intranasal antihistamine
Information on specific treatment options
  • Intranasal corticosteroid is the most effective treatment, but it has a slow onset of action – it may take hours to days for it to become effective
  • Antihistamines have a fast onset of action (within minutes), but are less effective than intranasal corticosteroids

2nd Line Therapy

Consider the following causes for treatment failure:

  • Compliance
  • Technique for using intranasal spray (the nozzle should be aiming for the lateral nasal wall, and the patient should just breathe in gently, instead of sniffing)
  • Alternative diagnosis

Consider the following further therapies:

Further therapy Indication  / description
Regular oral antihistamines (instead of PRN use) Especially if there is persistent nasal itching and sneezing
Intranasal anticholinergic (e.g. ipratropium bromide) Especially if there is persistent watery rhinorrhoea
Intranasal decongestant (e.g. xylometazoline) Especially if congestion is a problem

Only for short-term use (up to 5-7 days), as long-term use is associated with:

  • Rebound congestion upon withdrawal
  • Tolerance development
  • Nasal mucosa hypertrophy (‘rhinitis medicamentosa’), which further worsens the symptoms
LTRA (e.g. montelukast) If there is a history of asthma and ongoing symptoms
Oral corticosteroids “Last resort” due to side effects of systemic steroids

  • Ideally, only for adults
  • Short courses (5-10 days)
  • Only if there are severe, uncontrolled symptoms that are affecting quality of life

References

Related Articles

Sinusitis (Rhinosinusitis)

Nasal Polyps

Asthma (Chronic)

Allergic Conjunctivitis

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