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Juvenile Idiopathic Arthritis (JIA)

2026 American College of Rheumatology (ACR) Juvenile Idiopathic Arthritis (JIA) Guidelines.

Juvenile Idiopathic Arthritis (JIA)

Juvenile idiopathic arthritis (JIA) is an umbrella term for inflammatory arthritis of unknown cause that lasts for at least 6 weeks, with an onset before 16 y/o.

Causes and Risk Factors

The exact underlying cause remains unknown [Ref]

  • More common in females
  • Genetic factors play a major role

Clinical Features

The hallmark feature of JIA is chronic arthritis (lasting >6 weeks) in a child (<16 y/o) +/- extra-articular manifestations depending on the JIA subtype. [Ref]

Articular features Typically presents as:

  • Arthralgia
  • Synovitis
  • Joint effusion
  • Soft tissue swelling

Most commonly affected joints:

  • Wrists
  • Knees
  • Ankles

The number and pattern of joint involvement varies, depending on the subtype:

  • Oligoarticular pattern (<5 joints) – most common (~50-60% cases)
  • Polyarticular pattern (≥5 joints)
  • Systemic arthritis pattern
  • Enthesitis-related pattern (often with sacroiliac joint tenderness and/or inflammatory lower back pain)
  • Psoriatic pattern (often accompanied by dactylitis)

 

Extra-articular features Constitutional symptoms

  • Fever lasting >2 weeks – characteristic feature of systemic JIA
  • Generalised lymphadenopathy

Dermatological:

  • Salmon-coloured maculopapular rash
  • Psoriatic changes possible (e.g. psoriasis, nail changes)

Ophthalmological:

  • Uveitis (typically chronic anterior uveitis – which tends to be insidious and asymptomatic)

Other features:

  • Hepatomegaly and/or splenomegaly
  • Serositis – may manifest as pericarditis / pleuritis / peritonitis
  • Dactylitis
  • Enthesitis

Complications

Most common complications: [Ref]

  • Leg-length discrepancy
  • Joint contractures

Other complications: [Ref]

  • Growth retardation
  • Reduced bone mineral density
  • Hip involvement may require joint replacement
  • Amyloidosis
  • Interstitial lung disease

Macrophage activation syndrome is an extremely severe complication, suggested by:

  • Persistent high fever and CRP
  • Low ESR and fibrinogen
  • High or rising ferritin level (>1,000)
  • Cytopaenia (at least 2 cell lineages) or falling cell counts
  • High triglycerides and LFTs

Investigation and Diagnosis

JIA is a diagnosis of exclusion [Ref]

  • No single objective test can definitively diagnose JIA
  • A clinical diagnosis can only be made if there is chronic arthritis (>6 weeks) in a child (<16 y/o), after all other possible causes of chronic arthritis have been ruled out

Initial work-up for suspected JIA: [Ref]

  • FBC, ESR, CRP (would show signs of inflammation – e.g. ↑ ESR/CRP)
  • ANA
    • Used to exclude differential diagnoses like SLE, mixed connective tissue disease
    • +ve ANA is a major predictor and risk factor for developing chronic anterior uveitis [Ref]
  • Rheumatoid factor, anti-CCPHLA-B27
    • They provide limited diagnostic value
    • But they are mainly used to help classify the subtype, and their presence is associated with poor prognosis
  • Imaging
    • Initial test: X-ray (but cannot detect early JIA)
    • Ultrasound
    • MRI (most sensitive modality to detect early synovitis and demonstrate bone marrow oedema)

It is important to perform the following complication screening:

  • Ophthalmic screening for uveitis is strongly recommended (every 3 months)
    • JIA-associated chronic anterior uveitis is classically known to be insidious and asymptomatic
    • Therefore, frequent screening is the only way to detect uveitis before leading to sight-threatening complications
    • Note that +ve ANA is a major risk factor and predictor for the development of JIA-associated chronic anterior uveitis
  • Consider lung disease screening (for those with systemic JIA)

Management

The mainstay disease-modifying therapy for JIA is DMARDs:

JIA type Management
Non-systemic JIA 1st line:

  • Conventional DMARD (methotrexate is preferred)
  • +/- Biological DMARD (TNF-alpha inhibitor is preferred)

Adjuncts

  • Intra-articular steroid injection (triamcinolone hexacetonide) is strongly recommended for oligoarthritis
  • NSAIDs for symptomatic relief
Systemic JIA 1st line: biological DMARD

  • IL-1 inhibitor or IL-6 inhibitor
  • If the patient also has MAS, also give systemic steroids

Monotherapy of conventional DMARDs or NSAIDs is NOT recommended

Management of JIA-associated uveitis:

  • Topical steroid eye drops (prednisolone) for short-term management
  • DMARDs (methotrexate or adalimumab) are used to taper off steroid eye drops

References

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