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Placental Abruption

RCOG Antepartum Haemorrhage (Green-top Guideline No. 63). Last reviewed: Dec 2011.

Disclaimer:

This article is anchored to RCOG Green-top Guideline No. 63: Antepartum Haemorrhage (2011), which remains the current UK guideline for placental abruption in the absence of a dedicated NICE guideline. There is no standalone RCOG guideline specific to placental abruption.

Background Information

Definition

Placental abruption is the premature separation of the placenta from the uterine wall prior to delivery of the baby.

It involves bleeding from the site of placental separation within the uterus.

Epidemiology

Placental abruption is one of the most important causes of antepartum haemorrhage (bleeding from or into the genital tract occurring from 24 weeks of pregnancy up until the birth of the baby).

Antepartum haemorrhage complicates 3-5% of pregnancies and is a leading cause of maternal and perinatal mortality worldwide.

Placental abruption risk:

  • Baseline risk: 1.0%
  • Recurrent is 4.4% (after 1), and 19-25% (after 2)

Aetiology

The aetiology of placental abruption is not fully understood, but involves impaired placentation, placental insufficiency, uteroplacental underperfusion, and rupture of maternal decidual arteries, causing dissection of the decidual-placental interface. [Ref]

Risk Factors

~70% of all placental abruption cases occur in completely low-risk pregnancies. The majority of cases happen as a sudden and unexpected obstetric emergency without any known pre-existing conditions.

Risk factors for placental abruption include:

Category Specific risk factors
Lifestyle and environmental factors
  • Smoking
  • Drug misuse, specifically cocaine and amphetamines
  • Abdominal trauma (e.g. accidental trauma, domestic violence)
Obstetric and reproductive history
  • Abruption in a previous pregnancy – most predictive risk factor
  • Multiparity
  • Assisted reproductive technologies
Maternal characteristics
  • Advanced maternal age
  • Low BMI

Medical condition:

  • Pre-eclampsia
  • Thrombophilias (specifically factor V Leiden and prothrombin mutations)
Current pregnancy complications
  • 1st trimester bleeding
  • Intrauterine haematoma (identified on 1st trimester ultrasound)
  • Intrauterine growth restriction
  • Intrauterine infection
  • Polyhydramnios
  • Premature rupture of membranes
  • Non-vertex presentations

Clinical Features

Placental abruption is a cause of antepartum haemorrhage – bleeding that occurs from 24 weeks onwards

Sudden onset of:

  • Abdominal pain (continuous)
  • Vaginal bleeding (may be ‘concealed’ / not be present – see below)

Other signs:

  • Uterine sign
    • Woody or tense uterus on palpation
    • Uterine tenderness
  • Fetal compromise (on CTG – as it often causes fetal hypoxia)
  • Massive haemorrhage may cause maternal shock (e.g. tachycardia, hypotension, syncope)

~18% of placental abruption cases are concealed, where the bleeding is trapped behind the placenta, causing no or minimal vaginal bleeding. [Ref]

This is clinically important as the severity of haemorrhage cannot be reliably determined by the volume of external bleeding alone (visible blood loss can be little, but internal bleeding is significant).

Placental abruption should be suspected in the presence of continuous abdominal pain, a tense or woody uterus, and clinical shock, even in the absence of vaginal bleeding.

Complications

Maternal complications
  • Maternal shock
  • Consumptive coagulation (DIC)
  • ↑ Risk of postpartum haemorrhage
Fetal complications
  • Fetal hypoxia and anaemia
  • Preterm birth – either spontaneous (abruption precipitates labour) or iatrogenic (if early delivery is indicated)
  • Small for gestational age and fetal growth restriction – if the pregnancy continues after a mild abruption)

Diagnosis

Placental abruption is primarily a clinical diagnosis.

The following tests are recommended in patients with antepartum haemorrhage, but NOT to diagnose the abruption itself.

Blood Tests

Standard:

  • FBC
  • Group and save

For massive haemorrhage, also perform a coagulation screen, U&E, LFT, and cross-match

Imaging

1st line: ultrasound

  • Normal ultrasound does not exclude abruption because it has limited sensitivity (~24%) in identifying retroplacental haemorrhage
  • The primary purpose is to confirm or exclude placenta praevia (which can also cause antepartum haemorrhage)

Fetal Monitoring

Perform continuous CTG

  • Fetal well-being is important in guiding management

If the fetal heartbeat cannot be heard using external auscultation → ultrasound immediately to establish whether there is fetal heart pulsation and rule out fetal death

Management

Immediate Management

Immediate priority: resuscitation and stabilisation of the mother

The mother must be stabilised before any decision is made regarding the fetus or establishing the fetal condition.

Definitive Management

First, determine whether there is maternal or fetal compromise:

Maternal compromise Fetal compromise
Characterised by:

  • Life-threatening bleeding
  • Cardiovascular instability
  • Signs of clinical shock
Indicated by abnormal CTG, including:

  • Fetal tachycardia (>160 bpm) – early sign of fetal hypoxia
  • Fetal bradycardia (rate <110 bpm) – prolonged or severe bradycardia is a critical sign of acute fetal distress and profound hypoxia
  • Sinusoidal pattern – rare but strongly associated with fetal anaemia or severe hypoxia

Maternal and/or Fetal Compromise (ANY Gestation)

Immediate delivery (usually category 1 “crash” Caesarean section)

Maternal compromise is characterised by:

  • Life-threatening bleeding
  • Cardiovascular instability
  • Signs of clinical shock

Fetal compromise is evidenced by abnormal CTG features, especially:

  • Sustained fetal bradycardia (baseline heart rate <110 bpm)
  • Prolonged decelerations (>3 minutes)

Stable Mother and Fetus

Where there is no maternal or fetal compromise, management is guided by gestational age.

Gestation Recommended management
Term (≥37 weeks) Consider induction of labour
Pre-term (<37 weeks) Conservative management is appropriate (given the mother and fetus are stable)

  • Offer maternal corticosteroid if 24-34+6 weeks

In cases of extremely pre-term (<26 weeks), the decision is complex and senior obstetric input should be sought.

Intrauterine Fetal Death

Vaginal birth is the recommended mode of delivery – regardless of gestation

References

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