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Antiphospholipid Syndrome (APS)

BSH Guidelines on the investigation and management of antiphospholipid syndrome. Last reviewed: Aug 2025.

The article has been optimised and restructured to improve clarity and learning flow.

Date: 18/03/26

Antiphospholipid Syndrome (APS)

Definition

APS is an autoimmune pro-thrombotic disease characterised by:

  • Clinical evidence of thrombosis (arterial, venous, or small vessel) and/or pregnancy complications
  • In the presence of persistently +ve antiphospholipid antibodies

Causes and Risk Factors

APS could be primary or secondary: [Ref]

Primary Idiopathic
Secondary Occurs in association with other autoimmune diseases:

  • SLE – most common (~40% cases)
  • RA – (~20% cases)

Pathophysiology

Antiphospholipids are pro-thrombotic → leads to a hypercoagulable state and thrombosis

Clinical Manifestation

Young female with unprovoked thrombosis or recurrent miscarriage → consider APS

Possible clinical manifestations:

Category Clinical manifestation
Thrombotic manifestation Venous thrombosis (most common)

  • DVT / PE
  • Unusual site thrombosis (e.g. Budd-Chiari syndrome, portal vein thrombosis, cerebral venous sinus thrombosis, renal vein thrombosis)

Arterial thrombosis

  • Stroke / TIA (most common arterial thrombosis)
  • Myocardial infarction
  • Renal artery thrombosis
Obstetric manifestation
  • Recurrent miscarriage (<10 weeks gestation)
  • Unexplained fetal death (≥10 weeks gestation)
  • Pre-term birth due to eclampsia / pre-eclampsia / placental insufficiency
  • Fetal growth restriction from placental insufficiency
Other features
  • Livedo reticularis (classic skin sign)
  • Heart valve disease (Libman–Sacks endocarditis)
  • Migraines
  • Neurocognitive disorders

Catastrophic APS is a rare, life-threatening form of APS characterised by rapid, widespread small-vessel thrombosis, leading to multiorgan failure and requiring urgent treatment.

Diagnosis

APS Testing

Indications for testing ANY of the following:

  • All newly diagnosed SLE
  • Unprovoked venous thrombosis
  • <50 y/o with arterial thrombosis with no other vascular risk factors
  • Obstetric clinical criteria – any of the following (based on the assumption that it is NOT caused by fetal / parental anomalies):
    • ≥3 consecutive unexplained miscarriages <10 weeks
    • ≥1 unexplained fetal death ≥10 weeks
    • ≥1 premature birth <34 weeks due to eclampsia / pre-eclampsia / placental insufficiency
APS testing Test for:

  • Anticardiolipin antibodies (IgG or IgM)
  • Anti-beta-2-glycoprotein I antibodies (IgG or IgM)
  • Lupus anticoagulant

Do NOT routinely test for APS in patients with VTE provoked by a major transient risk factor (e.g. surgery, immobilisation) or in those with active cancer.

Diagnostic Criteria

Diagnostic criteria (revised Sapporo criteria):

  • At least 1 clinical criterion (vascular or obstetric event), and
  • Laboratory criterion
Criterion category Description
Clinical criterion Vascular thrombosis event

  • At least 1 objectively confirmed episode of arterial / venous / small-vessel thrombosis in any organ
Obstetric event (pregnancy morbidity) – any of the following (based on the assumption that it is NOT caused by fetal / parental anomalies):

  • ≥3 consecutive unexplained miscarriages <10 weeks
  • ≥1 unexplained fetal death ≥10 weeks
  • ≥1 premature birth <34 weeks due to eclampsia / pre-eclampsia / placental insufficiency
Laboratory criterion To meet this criterion: +ve antiphospholipid antibodies (any of the following) on 2 occasions at least 12 weeks apart

  • Lupus anticoagulant
  • Anticardiolipin antibody
  • Anti-β2 glycoprotein-1 antibody

Triple +ve APS

Defined as the persistent presence of 1) lupus anticoagulant, 2) anticardiolipin antibody, and 3) anti-β2 glycoprotein-1 antibody

Triple +ve APS is the highest-risk antibody profile, associated with:

  • Significantly higher risk of thrombotic and obstetric events
  • Poor responses to DOACs

Patients who meet the laboratory criteria but not the clinical criteria (i.e. those with antiphospholipid antibodies but no history of thrombotic or obstetric events) do NOT have antiphospholipid syndrome. Instead, they are described as asymptomatic antiphospholipid carriers.

Other Laboratory Findings

Haematological findings in APS (but not part of the diagnostic criteria): [Ref]

  • Thrombocytopaenia
  • Prolonged aPTT

Laboratory findings of SLE may also be present in secondary APS:

  • FBC: haemolytic anaemia, leukopaenia
  • Serology: +ve ANA, anti-dsDNA, anti-Smith antibodies
  • Low C3 and C4
  • ↑ ESR, normal CRP

Note: These findings are associated with APS with underlying SLE and are NOT typical of primary APS (or APS from other secondary causes).

See the Systemic Lupus Erythematosus (SLE) article for more information.

Management

Asymptomatic Antiphospholipid Carriers (Primary Prevention)

This refers to those with persistently positive antiphospholipid antibodies but no history of thrombotic or obstetric events

Management aspect Description
Cardiovascular risk factor management Primary management aim in primary prophylaxis

  • Lifestyle advice (including smoking cessation, regular physical activity, and weight loss)
  • Strict control of hypertension and diabetes
  • Manage hypercholesterolaemia
Low-dose aspirin Routine use of low-dose aspirin for primary prophylaxis is NOT recommended

Exception: consider in those who have had a history of obstetric events but never had a thrombotic event (after careful risk and benefit assessment)

Hydroxychloroquine Routine use of hydroxychloroquine for primary prophylaxis is NOT recommended

Exception: consider in those with triple +ve APS, esp. if additional vascular risk factors are present

Anticoagulation is generally NOT recommended for primary prophylaxis.

BSH guidelines noted a conflict with EULAR guidelines.

  • EULAR guidelines recommend low-dose aspirin in APS patients with high-risk profiles
  • However, BSH guidelines maintain a recommendation against routine use because prospective studies have failed to show a significant benefit for aspirin in reducing first-time thrombotic events

Established Thrombotic Antiphospholipid Syndrome (Secondary Prevention)

This refers to those with established antiphospholipid syndrome who had a previous thrombotic event +/- obstetric event.

After First Thrombotic Event

Thrombosis type Management
Venous thrombosis 1st line: warfarin

  • Target INR: 2.0-3.0
  • Duration
    • If unprovokedindefinite (lifelong)
    • If provoked → case-by-case (indefinite anticoagulation is likely necessary in those with a high-risk antibody profile like triple +ve)
Arterial thrombosis (stroke / TIA / MI) 1st line: warfarin

  • Target INR: 2.0-3.0
  • Duration: indefinite (lifelong)
  • If the patient has additional vascular risk factors and no significant risk of bleeding → consider adding an antiplatelet (e.g. aspirin) in addition to warfarin

If warfarin is contraindicated → consider dual antiplatelet therapy

When initiating warfarin, a heparin lead-in is required. A fast-acting anticoagulant (usually LMWH) is started alongside warfarin and continued until the INR reaches the target therapeutic range, usually 2.0-3.0. The heparin can then be discontinued, and the patient is maintained on warfarin alone.

DOACs (e.g. apixaban, rivaroxaban) should be avoided (esp. in triple +ve APS)  due to significantly higher risk of arterial thrombosis (esp. stroke), compared to warfarin.

If the patient is anticoagulated with DOACs, they should be switched to warfarin.

Recurrent Thrombosis Despite Anticoagulation

The following steps should be performed:

  • Specialist referral
  • Ensure the patient is on warfarin (if on DOAC → switch to warfarin)
  • Optimise warfarin therapy (e.g. review drug interactions, adherence, accuracy of INR measurement)

If the patient is experiencing recurrent thrombosis despite optimal anticoagulation with warfarin (INR 2.0-3.0), escalation of therapy is necessary:

  • Step 1:
    • Increase target INR to 3.0-4.0 (on warfarin), OR
    • Add an antiplatelet (aspirin) + maintain target INR of 2.0-3.0
  • Step 2: consider adding hydroxychloroquine
  • Step 3 (highly refractory cases): consider rituximab or complement inhibitors (e.g. eculizumab)

Obstetric Antiphospholipid Syndrome (Management in Pregnancy)

Medication change upon confirmation of pregnancy
  • Stop warfarin immediately (as it is teratogenic)
  • Give low-dose aspirin + LMWH throughout pregnancy to prevent obstetric events
Fetal monitoring to detect signs of placental dysfunction
  • Uterine artery Doppler scan is recommended at 20-24 weeks gestation (strong predictor of pre-eclampsia and fetal growth restriction)
  • If the Doppler is abnormal → perform serial growth scans to monitor for fetal growth restriction

Also see the Small for Gestational Age (SGA) and Fetal Growth Restriction (FGR) and Hypertension in Pregnancy (Gestational Hypertension, Pre-Eclampsia, and Eclampsia) articles

Catastrophic Antiphospholipid Syndrome

Catastrophic APS is a medical emergency that requires a multidisciplinary approach involving haematologists, intensive care clinicians and other relevant specialists.

Acute management involves:

  • 1st line: triple therapy with therapeutic dose IV heparin + high-dose corticosteroid + IVIG and/or plasma exchange
  • 2nd line: consider cyclophosphamide, rituximab, eculizumab

References

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