Disclaimer
We’re actively expanding Guideline Genius to cover the full UKMLA content map. You may notice some conditions not uploaded yet, or articles that only include diagnosis and management for now. For updates, follow us on Instagram @guidelinegenius.
We openly welcome any feedback or suggestions through the anonymous feedback box at the bottom of every article and we’ll do our best to respond promptly.

Thank you for your support.
The Guideline Genius Team

Total Live Articles: 326

Multiple Myeloma (MM)

NICE guideline [NG35] Myeloma: diagnosis and management. Last updated: Oct 2018.

NICE guideline [NG12] Suspected cancer: recognition and referral 1.10 Haematological cancers. Last updated: Jan 2026.

The article has been updated in line with NICE guideline.

Date: 23/02/26

Background Information

Definition

MM is a haematologic malignancy defined by the presence of abnormal plasma cells within the bone marrow.

Aetiology

Epidemiology: [Ref]

  • Median age at onset: 69 y/o
  • More common in black individuals (2x compared to white individuals)
  • More common in males

 

Risk factors: [Ref]

  • Obesity
  • MGUS (nearly all cases of MM are preceded by MGUS)

Diagnosis

Approach:

  • MM should be suspected in >60 y/o patients with persistent bone pain (esp. back pain) or unexplained fracture
  • If MM is suspected, offer the following initial investigations:
    • FBC
    • Calcium level
    • ESR
    • Serum protein electrophoresis
    • Serum free light chain (if not available → urine electrophoresis)
  • If the initial investigations are suggestive of MM → suspected cancer pathway referral.
  • Secondary care investigations would include:
    • Imaging
    • Definitive test: bone marrow analysis

Clinical Manifestation (End-Organ Damage)

The CRAB criteria represent the classic manifestation of end-organ damage caused by MM: [Ref]

Calcium high Features of hypercalcaemia:
  • Renal stones: renal / ureteric stones, polyuria and polydipsia (from nephrogenic diabetes insipidus)
  • Painful bones: bone / muscle / joint pain, pseudogout, muscle weakness
  • Abdominal groans: abdominal pain, constipation, anorexia, N&V, pancreatitis
  • Psychic moans: depression, fatigue, confusion
Renal impairment Often asymptomatic, detected on blood tests

Late manifestations include oliguria / anuria, fluid overload, uraemic symptoms

Anaemia Features of anaemia
  • Fatigue and reduced exercise tolerance
  • Exertional dyspnoea
  • Pallor
  • Dizziness / lightheadedness
  • Palpitations
Bone lesions Bone lesions in MM include:
  • Bone pain (classically back pain)
  • Pathological fractures
  • Vertebral collapse / fracture → height loss, kyphosis, cord compression

Manifestations beyond the CRAB criteria: [Ref]

  • Thrombocytopenia → bleeding / bruising
  • MM-related immunodeficiency (B-cell and T-cell dysfunction) → recurrent infections (esp. chest infection)

10-15% of patients with MM have concurrent light chain amyloidosis, which can cause:

  • Macroglossia
  • Periorbital ecchymoses (panda eyes)
  • Submandibular gland enlargement
  • Unexplained cardiomyopathy

Laboratory Tests

[Ref]

FBC
  • Anaemia – normocytic, normochromic (most common)
  • Thrombocytopaenia
  • Leukopaenia
ESR
  • ↑ (due to increased plasma immunoglobulins, which promote erythrocyte aggregation)
Bone profile
  • Hypercalcaemia with normal ALP
U&E and metabolic panel
  • Renal impairment (↑ serum creatinine, ↓ eGFR)
  • ↑ ESR (classically very high)
  • ↑ Uric acid (high cell turnover)
Peripheral blood smear
  • Rouleaux formation

Protein Studies

There are 3 main protein studies: [Ref]

Serum free light chain assay
  • ↑ Free κ or ↑ free λ light chains
  • Abnormal κ:λ ratio
  • ↑ Involved:uninvolved light chain
Serum protein electrophoresis
  • Monoclonal (“M”) protein spike (most commonly IgG, then IgA)
  • Immunoparesis (↓ normal polyclonal immunoglobulins)
  • ↑ Total protein / globulins
24-Hour urine electrophoresis
  • Bence Jones proteinuria (monoclonal light chains in urine)

Imaging

Imaging should be offered to ALL patients with suspected MM:

  • 1st line: whole body MRI
  • 2nd line: whole body low-dose CT
  • 3rd line: skeletal survey (less sensitive – a lytic lesion only becomes visible on a standard X-ray after 30% to 50% of the bone cortex has been eroded)

 

Typical imaging findings include: [Ref]

  • Osteolytic lesions (at least 1 osteolytic lesion is needed to meet the diagnostic criteria)
  • Pathological fractures (esp. vertebrae, ribs)
  • Vertebral collapse / compression fractures

Bone Marrow Analysis

Definitive test: bone marrow aspirate + biopsy [Ref]

  • ≥10% of bone marrow plasma cells meet the diagnostic criteria

Management

Definitive Management

The first step is to determine if the patient meets the haematopoietic stem cell transplant eligibility, key criteria include: [Ref]

Adequate organ function
  • Severe cardiac, pulmonary, or hepatic disease is usually a contraindication
  • However, renal impairment or even renal failure does not necessarily preclude transplantation
Acceptable functional status
  • Including the ability to perform activities of daily living, indicating they can withstand intensive treatment and recovery
  • Consider frailty and performance status
Age consideration
  • Advanced age alone is not an absolute contraindication
  • However, most transplant recipients are typically under 70–75 y/o

Transplantation is intensive and generally unsuitable for patients with severe irreversible non-renal organ disease, poor functional status, or significant frailty.

Transplant Eligible

Standard 1st line treatment in transplant-eligible patients is made up of a 3-phase approach:

Phase 1: induction therapy NICE recommends bortezomib + dexamethasone +/- thalidomide

Usually given for 3-6 months to rapidly reduce tumour burden in the bone marrow

Phase 2: autologous haematopoietic stem cell transplant A 3-phase process:
  1. Stem cells mobilised and collected
  2. High-dose melphalan (chemotherapy) is given
  3. Stem cells are reinfused to reconstitute bone marrow function
Phase 3: maintenance therapy Low-dose lenalidomide monotherapy

Transplant Ineligible

Mainstay of management is systemic combination therapy:

  • 1st line: thalidomide + alkylating agent (cyclophosphamide / melphalan) + corticosteroid
  • 2nd line: bortezomib + melphalan + prednisolone

Complication Prevention / Management

Complication Prevention / management Indication
Bone disease 1st line: zoledronic acid (bisphosphonate) Indicated for all patients to reduce risk of fractures and bone disease
Renal disease Bortezomib + dexamethasone Newly diagnosed, myeloma‑induced acute renal disease
Infection Offer:
  • Seasonal influenza vaccination

Consider:

  • Extending pneumococcal vaccination to those who are <65 y/o
  • IVIV replacement in those with hypogammaglobulinaemia and recurrent infections
  • Aciclovir prophylaxis in those who take both immunomodulatory drugs and high-dose steroids
  • Testing for HIV, Hep B, Hep C before starting treatment

References

Share Your Feedback Below

UK medical guidelines made easy. From guidelines to genius in minutes!

Quick Links

Cookie Policy

Social Media

© 2026 GUIDELINE GENIUS LTD