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Ascending Cholangitis

Gallstone Disease Overview

Spectrum of Gallstone Disease Presentation

Be aware that gallstone disease can present as ANY of the following:

Condition Pathophysiology Clinical and biochemical manifestations Key investigations Management
Cholelithiasis Presence of gallbladder stone
  • Asymptomatic, or
  • Biliary colic
  • Ultrasound
  • Normal WCC and CRP
Elective laparoscopic cholecystectomy if symptomatic
Choledocholithiasis Presence of common bile duct stone
  • Obstructive jaundice
  • Abdominal pain
  • Ultrasound
  • Deranged LFT (cholestatic pattern)
  • Normal WCC and CRP
Bile duct clearance + laparoscopic cholecystectomy
Acute cholecystitis Persistent cystic duct obstruction → gallbladder inflammation
  • RUQ pain
  • Murph’s sign +ve
  • Fever
  • Ultrasound
  • ↑ WCC and CRP
  • Normal / mildly deranged LFT (no cholestasis)
IV fluids + antibiotics → early laparoscopic cholecystectomy
Acute cholangitis Common bile duct obstruction → ascending bacterial infection Charcot’s triad:

  • RUQ pain
  • Fever
  • Jaundice
  • MRCP
  • ↑ WCC and CRP
  • Deranged LFT (cholestatic pattern)
IV antibiotics + urgent ERCP
Gallstone pancreatitis Stone impacts at ampulla of Vater → pancreatic duct obstruction → pancreatitis
  • Epigastric pain radiating to the back
  • Nausea and vomiting
  • Ultrasound
  • ↑ Amylase / lipase
  • Deranged LFT (cholestatic pattern)
Supportive (IV fluids, analgesia, optimise nutrition) → cholecystectomy after recovery

Background Information

Definition

Ascending cholangitis is defined as a bacterial infection of the biliary tree, that occurs secondary to biliary obstruction

Aetiology and Pathophysiology

Most common cause of biliary obstruction in ascending cholangitis: [Ref]

  • Choledocholithiasis (common bile duct stone) – most common
  • Obstructing mass (e.g. cholangiocarcinoma, pancreatic adenocarcinoma)
  • Benign or malignant biliary strictures
  • Biliary stents
  • Mirizzi syndrome (obstruction of the common hepatic duct due to extrinsic compression from a gallstone impacted in the cystic duct or gallbladder neck, often accompanied by local inflammatory changes)

 

Pathophysiology: [Ref]

  • Biliary obstruction → bacteria ascend from the duodenum into the biliary system → colonisation and infection
  • Most common organism: gram -ve enteric bacteria (e.g. E. coli, Klebsiella species)

Risk Factors for Gallstone Formation

Risk factors for gallstone formation can be divided according to the composition of the gallstone: [Ref]

Cholesterol gallstone (most common)  5 Fs

  • Female (oestrogen increases cholesterol secretion in bile)
  • Fat (obesity / metabolic syndrome)
  • Forty (middle-aged)
  • Fertile (pregnancy / oestrogen therapy – due to state of increased oestrogen)
  • Fair skin (caucasian ethnicity)

 

Other risk factors

  • Ileal disease (e.g. Crohn’s disease) or ileal resection
  • Cirrhosis (due to ↓ bile acid synthesis → ↑ relative cholesterol secretion)
  • Drugs
    • Oestrogen-containing drugs (e.g. COCP, HRT)
    • Fibrates
    • GLP-1 agonists (e.g. liraglutide)
Pigmented gallstone Causes of chronic haemolysis

  • Sickle cell disease
  • Hereditary spherocytosis
  • Other causes of haemolytic anaemia

Other causes

  • Cirrhosis
  • Chronic biliary tract infection or stasis

Complications

Key complications:

  • Sepsis → septic shock → multiple organ dysfunction syndrome (esp. cardiovascular, renal and respiratory failure) – most important
  • Pyogenic liver abscess
  • Pericholecystic abscess
  • Biliary strictures (may cause biliary cirrhosis from prolonged obstruction and infection)

Diagnosis

Diagnostic criteria: [Ref]

  • Typical clinical features, and
  • Systemic signs of inflammation, and
  • Signs of cholestasis, and
  • Confirmatory imaging findings

Clinical Features and Blood Tests

Typical textbook presentation (not always present): [Ref]

  • Charcot’s triad (high specificity but poor sensitivity): fever + jaundice + RUQ pain
  • Reynold’s pentad: Charcot’s triad + hypotension + altered mental status

 

Features of the diagnostic criteria: [Ref]

Systemic signs of inflammation
  • Fever
  • ↑ White cell count
  • ↑ CRP
Signs of cholestasis
  • Jaundice
  • ↑ Bilirubin level
  • Cholestatic LFT derangement (↑ ALP and GGT)

The classic table of biliary colic vs acute cholecystitis vs ascending cholangitis:

Condition Pain (RUQ / epigastric) Signs of inflammation (fever / ↑ WCC / ↑ CRP) Signs of biliary obstruction (jaundice / hyperbilirubinaemia)
Cholelithiasis (biliary colic) Yes No No
Choledocholithiasis Yes No Yes
Acute cholecystitis Yes Yes No
Ascending cholangitis Yes Yes Yes

Imaging

1st line: abdominal ultrasound [Ref]

  • Findings:
    • Biliary duct dilatation
    • Evidence of an underlying cause (e.g. stone, stricture, obstructing mass)

2nd line: MRCP / CT abdomen with IV contrast

  • Consider if abdominal ultrasound has not detected common bile duct stones BUT suspicion remains

Management

Initial Management

Initial management involves supportive care: [Ref]

  • IV fluid resuscitation as appropriate
  • Empirical antibiotics (targeting enteric gram-negative and anaerobic organisms)

Definitive Management

Definitive management: biliary decompression via ERCP [Ref]

  • Ideally it should be performed when patients are hemodynamically stable (after initial resuscitation)

Technically, ERCP is not routinely indicated in all patients with ascending cholangitis

  • Mild (grade I) disease does NOT routinely require ERCP if there is improvement with supportive care and antibiotics. ERCP is reserved for those failed to respond to medical therapy
  • Otherwise, non-mild disease (grade II-III) requires routine ERCP (urgency depends on severity)

However, exam questions typically expect one to choose ERCP as the “most appropriate management”. Unless the scenario specifies that the patient is responding well to medical therapy and declines any invasive intervention.

Secondary Prevention

Secondary prevention is achieved via prophylactic cholecystectomy [Ref]

  • It is offered after an episode of gallstone-related cholangitis in patients who are clinically stable AND have an acceptable surgical risk
  • NOT routinely offered to patient with non-gallstone (i.e. acalculous) aetiologies

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