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Peripheral Arterial Disease (PAD)

NICE clinical guideline [CG147] Peripheral arterial disease: diagnosis and management. Last updated: Dec 2020

Minor changes and restructuring to the ‘Definition’ and ‘Clinical Features’ sections have been made.

Date: 13/11/25

Background Information

Definitions

PAD is defined by the presence of atherosclerotic obstruction in the peripheral arteries of the lower limb

 

The clinical presentation of PAD can be categorised into 4 subsets: [Ref]

  • Asymptomatic
  • Chronic symptomatic PAD
  • Chronic limb-threatening ischaemia (old term: critical limb ischaemia)
  • Acute limb ischaemia: sudden (<2 weeks) decrease in limb perfusion that threatens limb viability (covered in a separate article)

Clinical Features

Patients with PAD often have concomitant cardiovascular risk factors:

  • Smoking
  • Diabetes
  • Hyperlipidaemia
  • History of coronary artery disease / cerebrovascular disease

 

There are 3 main clinical manifestations of chronic PAD: [Ref]

Presentation Symptoms Signs
Asymptomatic Note that these patients may self-limit and adapt their activity to remain below their ischemic threshold to avoid leg pain
  • Diminished or absent lower limb pulses
  • Mild trophic changes (e.g. hair loss, thin skin)
Chronic symptomatic PAD Most commonly presents as intermittent claudication (“angina of the legs”):
  • Lower limb pain that is brought on after walking a predictable distance
  • Relieved by rest
  • NO resting leg pain

 

Location of pain:

  • Aortoiliac disease (Leriche syndrome) → triad of buttock / hip / thigh claudication + erectile dysfunction + absent / diminished femoral pulses
  • Femoropopliteal disease → calf claudication (most common)
  • Infrapopliteal disease → foot claudication
Chronic limb-threatening ischaemia (critical limb ischaemia – old term) Characterised by the presence of
  • Lower limb pain at rest (typically affects toes and forefoot first)
  • Pain is worse at night (patients may report hanging the leg out of bed / sleeping in a chair to relieve pain)
Characterised by the presence of tissue loss:
  • Non-healing arterial ulcer
  • Gangrene (usually dry gangrene)

Other findings:

  • Skin pallor on limb elevation (+ve Buerger sign)
  • Cool skin
  • Markedly delayed capillary refill (>5 sec)
  • Dry and shiny skin

 

Key exam presentations:

  • Pain with walking that is relieved by rest = intermittent claudication
  • Pain at rest +/- ulcer or gangrene = chronic limb-threatening ischaemia (critical limb ischaemia)

Guidelines

Investigation and Diagnosis

Approach:

  • 1st line: ABPI (in clinic)
  • Then, perform imaging in secondary care if revascularisation is being considered

 

ABPI (Ankle Brachial Pressure Index)

How to measure:

  • Similar to measuring a clinic BP – instead of using a stethoscope, a handheld doppler is used
  • BP cuff placed on the arm and ankle
  • Use doppler to locate the pulse (DP, PT pulses & brachial pulses)
  • Inflate the cuff until the pulse is no longer audible on doppler
  • Deflate the cuff slowly and note the pressure (systolic BP) when doppler is audible again

ABPI calculation: highest ankle systolic BP (DP or PT) / highest brachial systolic BP (right or left arm)

Interpretation:

ABPI Interpretation
>1.4 May suggest arterial calcification/stiffness (typically diabetes)
Unable to rule in or out PAD
1.0 – 1.4 Normal
≤0.9 PAD
<0.5 Chronic limb-threatening ischaemia

Do not exclude a diagnosis of peripheral arterial disease in people with diabetes based on normal or raised ABPI alone.

  •  Diabetes can cause arterial calcification, which makes arteries more incompressible, thus a falsely high ABPI reading.

Alternative diagnostic test if ABPI is >1.4 (suggesting arterial calcification/stiffness) →  toe-brachial index (TBI) [Ref]

  • Same as ABPI but using digital artery pulse in the toes.

 

Imaging

Imaging should be performed if revascularisation is being considered:

  • 1st line: duplex ultrasound
  • 2nd line: MR angiography with contrast
  • 3rd line: CT angiography

Management

Definitive Management

Intermittent Claudication

Step 1 Supervised exercise programme
Step 2 Revascularisation (see below for choosing approaches)
  • Endovascular approach – angioplasty + stenting
  • Open approach – bypass surgery
Step 3 (considered if surgery is inappropriate for patient) Naftidrofuryl oxalate

NICE did not make any specific recommendations on choosing between endovascular vs open revascularisation approaches. Selected points from the 2024 ACC/AHA guidelines: [Ref]

Endovascular revascularisation (angioplasty + stenting) is generally 1st line.

Open revascularisation is preferred as 1st line in

  • Multilevel occlusions
  • Long-segment (>10cm) occlusions
  • Lesions involving the common femoral artery and profunda femoris artery origin

Chronic Limb-Threatening Ischaemia (Critical Limb Ischaemia)

  • Refer all patients to vascular specialist
  • Offer revascularisation to all patients
    • Recent trials suggest that open revascularisation is superior to endovascular in those with chronic limb-threatening ischaemia, especially if there is a suitable autogenous vein (e.g. great saphenous vein)

Secondary Prevention

Offer all the following:

  • Lifestyle changes + treat comorbidities
  • Atorvastatin 80mg PO OD
  • Clopidogrel 75mg PO OD

References

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