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Necrotising Enterocolitis (NEC)

NHS GGC Necrotising enterocolitis in Paediatric Intensive Care, management guideline (1186). Last reviewed: Jan 2025.

Necrotising Enterocolitis (NEC)

NEC is a haemorrhagic necrotising inflammation of the intestinal wall that most commonly affects the distal ileum and proximal colon

Causes and Risk Factors

Primary risk factor: prematurity [Ref1][Ref2]

  • Preterm infants have immature GI tracts with poor peristalsis, high mucosal permeability, and reduced secretion of digestive enzymes and protective gastric acid

Other risk factors: [Ref1][Ref2]

  • Low birth weight
  • Formula milk feeding
  • Prolonged empirical antibiotic exposure
  • Reduced intestinal perfusion (e.g. congenital heart disease)
  • Gastric acid-suppressing medications (PPIs or H2 antagonist)

Human breast milk feeding is protective against NEC [Ref1][Ref2]

Clinical Features

[Ref1][Ref2]

Local GI features
  • Feeding intolerance +/- vomiting
  • Abdominal distension + tenderness
  • Reduced / absent bowel sounds
  • Bloody stools (haematochezia)
Systemic features
  • Temperature instability (hypothermia / pyrexia / temperature fluctuation)
  • Cardiovascular instability
  • Respiratory distress
  • Neurological / behavioural changes (e.g. lethargy, irritability, hypotonia)

Features of severe NEC: [Ref1][Ref2]

  • Abdominal wall erythema / discolouration / oedema
  • Shock
  • Respiratory failure

Investigations and Diagnosis

Blood Tests

Key tests and possible findings in NEC: [Ref1][Ref2]

Blood test Finding and/or rationale in NEC
FBC Neutrophilia may occur

Thrombocytopenia and neutropenia are associated with more severe disease

U&Es Assess electrolyte disturbances, including hyponatraemia, and renal function
Blood gas Metabolic acidosis and ↑ lactate may suggest tissue hypoperfusion or bowel ischaemia
Inflammatory markers ↑ CRP +/- ↑ procalcitonin
Coagulation screening May show coagulopathy, including DIC in advanced disease
Blood cultures To identify infections and guide antibiotic treatment

Blood cultures should be obtained before starting antibiotics, if possible (without delaying treatment)

Imaging

Diagnostic test of choice: abdominal X-ray (AXR) [Ref1][Ref2]

The classic 4 AXR finding in NEC: [Ref1][Ref2]

Staging (Bell’s) Radiographic finding Description
I Bowel dilatation / ileus
  • Asymmetrical, gas-filled, or dilated loops of bowel
  • Bowel wall thickening
  • Absence of colonic gas
II Pneumatosis intestinalis – pathognomonic sign of NEC Intramural gas (radiolucent lines outlining the intestinal wall)
Portal venous gas Branching dark lucencies over the liver (indicating gas within the portal venous system within the liver)
III Pneumoperitoneum Free gas within the peritoneal cavity, such as:

  • Free air under the diaphragm on an erect film
  • Double-wall sign (Rigler’s sign) – gas outlines both the inner and outer surfaces of the bowel wall
  • Football sign – a large oval area of lucency outlining the abdominal cavity
  • Falciform ligament sign – free gas outlines the falciform ligament, making it visible as a linear opacity

NEC Staging

Deitch-modified Bell staging for NEC: [NHS GGC]

Stage Classification Clinical features Radiological features
I Suspected necrotising enterocolitis
  • Abdominal distension
  • Bloody stools
  • Emesis
  • Apnoea
  • Lethargy
  • Bowel dilatation
  • Ileus
II Proven necrotising enterocolitis
  • Abdominal tenderness
  • +/- Metabolic acidosis, thrombocytopaenia
  • Pneumatosis intestinalis, and/or
  • Portal venous gas
III Advanced necrotising enterocolitis
  • Hypotension
  • DIC
  • Neutropaenia
  • Pneumoperitoneum

Complications

Acute complications: [Ref1][Ref2]

  • Intestinal perforation → peritonitis → sepsis → septic shock → death
  • NEC-induced acute lung injury
  • Severe metabolic and haematological derangements
    • Lactic acidosis
    • Thrombocytopaenia
    • Neutropaenia

Long-term complications: [Ref1][Ref2]

  • Short bowel syndrome and intestinal failure (in those requiring surgery) → parenteral nutrition dependency
  • Intestinal strictures / stenosis / fistulas
  • Neurodevelopmental disorder and cognitive deficit (as systemic inflammation disrupts the BBB)

Management

Approach:

  • ALL patients → conservative / medical management
  • Selected patients → surgery

Conservative / Medical Management

Bowel rest [Ref1][Ref2]
  • Discontinuation of enteral feeding (including breastfeeding, bottle feeding, NG/OG feeding) (NBM)
  • Nasogastric decompression
  • Total parenteral nutrition +/- IV fluids
Broad-spectrum antibioticsantifungal [NHS GGC]
  • Classic triple therapy (to cover gram +ve, gram -ve and anaerobes): ampicillin + gentamicin + metronidazole [Ref]
  • Duration ranges from 7-21 days (depending on severity)
Systemic support [Ref1][Ref2]
  • Correct fluid and electrolyte deficits
  • Cardiovascular (e.g. fluids, vasopressors) and respiratory support (e.g. oxygen, ventilation) if necessary

Surgical Management

Do NOT routinely offer surgical management to ALL patients with NEC.

It is reserved for severe NEC or those refractory to conservative management.

Indications for surgery [Ref1][Ref2] Absolute indication: pneumoperitoneum on abdominal X-ray

Relative indications:

  • Failed to improve with conservative management
  • Abdominal wall erythema / discolouration / severe distension (suggests underlying peritonitis)
  • Persistent metabolic derangements (e.g.  metabolic acidosis, neutropaenia, thrombocytopaenia)
  • Cardiorespiratory collapse (e.g. refractory hypotension, worsening respiratory failure, ventilatory requirement)
Surgical options [Ref1][Ref2] Primary option: laparotomy (necrotic bowel resection followed by stoma creation or primary anastomosis)

If unfit for surgery (e.g. extremely low birth weight, unstable for surgery) or as a temporary measure: primary peritoneal drainage

Antenatal corticosteroids in pre-term delivery reduces risk of NEC, beyond its benefit in reducing NRDS and IVH. [Ref]

References

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