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Hypernatraemia

Background Information

Definition

Hypernatraemia is defined as serum sodium >145 mmol/L

 

Severe hypernatraemia is generally defined as >160 mmol/L

Clinical Features

Key features: [Ref]

Mild to moderate hypernatraemia
  • Signs of dehydration – common (e.g. dry mucosa, reduced skin turgor, tachycardia, orthostatic hypotension, prolonged capillary refill)
  • Weakness, lethargy
  • Irritability, restlessness
  • Mild confusion
Severe hypernatraemia (>160 mmol/L)
  • Profound confusion
  • Somnolence
  • Stupor
  • Coma
  • Seizures
  • Cerebral haemorrhage or vascular rupture in extreme cases (due to brain shrinkage)

Sodium disturbances (hypernatraemia or hyponatraemia) do NOT typically cause ECG changes or arrhythmias.

Unlike abnormalities in potassium, calcium, or magnesium, which directly affect cardiac electrophysiology.

Chronic hypernatraemia is often asymptomatic, esp. in elderly patients. [Ref]

Hypernatraemia in infants can cause hyperpnoea, muscle weakness, restlessness and a characteristic high-pitched cry. [Ref]

Diagnosis

Diagnostic Approach

Key tests to order

  • Paired serum and urine osmolality
  • Urine sodium

 

Other tests:

  • Serum glucose
  • U&E
  • Serum sodium

Hypernatraemia always gives a hypertonic state (unlike hyponatraemia, which can be hypotonic / isotonic / hypertonic):

  • ↑ Serum osmolality
  • ↑ Haematocrit

Identifying the Underlying Cause of Hypernatraemia

Urine osmolality is the primary discriminator:

  • Low urine osmolality: intra-renal loss (kidney is not concentrating urine)
  • High urine osmolality: extra-renal loss (kidney is concentrating urine maximally)
Intra-renal loss causes
  • Diabetes insipidus (see the Diabetes Insipidus (DI) article for more information)
  • Osmotic diuresis (e.g. hyperglycaemia, mannitol)
  • Loop diuretics
  • Hyperaldosteronism and Cushing’s syndrome
Extra-renal loss causes Essential causes of water loss (not enough input or too much output):
  • GI losses (e.g. vomiting, diarrhoea, nasogastric tube, stoma)
  • Skin losses (e.g. burns, excessive sweating)
  • Decreased fluid intake
  • Impaired access to water
  • Insufficient fluid prescription
  • Hypertonic fluids

The causes of hypernatraemia can be further categorised based on fluid status and urine sodium, but are not included here as they are less clinically relevant and exam-important (as opposed to hyponatraemia).

Management

Treat the underlying cause once identified.

Mild Hypernatraemia

Replace missing water with: [Ref]

  • Oral water (NOT electrolyte drinks), or
  • IV glucose 5%

IV glucose (e.g., glucose 5%) is preferred over normal saline for correcting hypernatraemia. Because once the glucose is metabolised, it provides free water.

In contrast, normal saline is isotonic to slightly hypertonic and therefore does not lower serum sodium.

Severe Hypernatraemia (>170 mmol/L)

Give IV glucose 5% [Ref]

  • Only give IV sodium chloride 0.9% if the patient is volume-depleted and hypotensive

IV glucose (e.g., glucose 5%) is preferred over normal saline for correcting hypernatraemia. Because once the glucose is metabolised, it provides free water.

In contrast, normal saline is isotonic to slightly hypertonic and therefore does not lower serum sodium.

It is important NOT to correct hypernatraemia too quickly, due to the risk of cerebral oedema (rapid decrease of serum osmolality will cause water shifting into the brain cells rapidly, causing swelling)

To prevent cerebral oedema from occurring:

  • Maximum correction rate is 10 mmol/L per day

On the other hand, if hyponatraemia is corrected too rapidly, there is a risk of developing osmotic demyelination syndrome (rapid increase in serum osmolality will cause water shifting out of brain cells rapidly).

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